Facebook | CCSVI in Multiple Sclerosis: News from Dr. Zamboni - CCSVI lesions classified as congenital

Common sense always prevails.

Committee of experts from 47 countries and chaired by prof. Byung Lee B, Georgetown officially clasified CCSVI as congenital deformities, and prior venous lesions in MS.

http://phleb.rsmjournals.com/cgi/reprint/22/6/249.pdf

Report



But what's all the excitement about something everyone knew. It was obvious that CCSVI was present before or at the time of ms. No one can deny this.

But what causes the CCSVI? I would imagine it is not present from birth, so what has caused this to develop?

What alters the pressure inside those veins to cause them to twist and strangulate?

Answer based on those cases we see in thisisms who have adopted the Inclined Therapy method must be posture related!

How else can these people be recovering function and sensitivity without surgery?

More to the point, it certainly looks like that old favourite of humans, sleeping flat is suspected to be the main contributing factor for both ccsvi and ms.

Remember, this is the third study we are seeing identical patterns of recovery in.

And then those varicose veins recovering again without surgery using I.T. paints a glorious picture of how sleeping flat must have been the main contributing factor that initiated their development and maintained their progress. Again no surgery required!


So if this can happen in Varicose Veins, Chronic Venous Insufficiency and lead to recovery from multiple sclerosis, Parkinson's disease, spinal cord injury and "cerebral palsy in a child in Kent" it must be having an observable affect on CCSVI if CCSVI is contributing to ms. If CCSVI is not affected by I.T. then there is obviously another underlying cause.


And I suspect that the liquid crystal properties of myelin might have something to say about this.

Lesions are lesions. M.S. = Multiple scars in the nervous system and / or brain. Those scars have not gone away because someone has opened up the plumbing and placed an insert inside.

The circulation in the arteries and veins is separate from the nervous system. The heart does not affect the circulation in the nervous system, so this plumbing job can address lethargy and blood flow related problems, assisting people to become more active, which will inevitably lead to better posture for longer periods. Perhaps it is activity that is helping ms symptoms rather than the plumbing job?

But at least CCSVI now has an official stamp, it's a start.

YouTube - Bricks under the bed, Rat Poison and CCSVI

Experts: Sitting too much could be deadly - Boston.com

It's about time "the experts caught up" I have been researching this for 16 years and proven that posture is very important for maintaining circulation! Without circulation we are dust.

My research involves density changes in fluids from exhaling and constant water vapour loss from the skin and eyes.

Titlting the bed allows gravity to influence the density changes in the blood and in doing so assists the circulation.

Inclined therapy is where a person raises the head of the bed by 6 inches so the whole bed tilts from head to toe.

Andrew K Fletcher http://www.mstrust.org.uk/downloads/tips.pdf

Poor Postural Advice for people with ms.

Tips & Advice...

• Try to ensure that you get enough sleep - take naps during

the day if necessary. Give yourself permission to rest and put

your feet up whenever possible

• When you feel tired or worn out, stop what you are doing

and have a... rest before you start again - pace yourself

• Don't stand when you can sit down, and don't sit down

when you can lie down

• Be aware of your physical limitations and listen to your body

when it tells you to stop

• You might find that relaxation tapes and gentle music help

you to wind down

• If you know that you have a big event coming up that might

tire you out, make sure that you prepare for it, by getting as

much rest as possible beforehand


Flat bed rest advocated by an ms charity as the panacea for multiple
sclerosis. Yet proven to be a powerful degenerative influence on
healthy people, let alone some one with a neurological condition. There
are a huge amount of publications identifying flat bed rest as anything
but a safe and healthy sanctuary?


How many people with ms have read this harmful information and followed the advice only to find they have deteriorated rapidly?

People need to wake up to Inclined Therapy!

http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2457047/pdf/brmedj06852-0012.pdf

History Of Postural Paralysis. This document from The British Medical Journal, Published Augus 8th 1938 is very interesting, and confirms the importance of posture for neurological function. It is a must read for anyone using or considering Inclined Therapy.
Print and study thoroughly.

Plain Text Version

PARALYSIS DUE TO POSTURE
BY
E. B. CLAYTON, M.B., B.Ch.
DIRECTOR OF PHYSICAL TREATMENT DEPARTMENT, KING'S COLLEGE
HOSPITAL
The following cases of nerve paralysis, which are apparently
due to pressure during the maintenance of some
posture, have been collkcted from the notes of patients
attending the Physical Treatment Department of King's
College Hospital during the last ten years.
Anterior Tibial Nerve Paralysis
Case 1.-In a woman, aged 59, there was a sudden onset
of foot-drop when she was kneeling to clean a grate in April,
1926. On examination the external popliteal nerve was
found to be tender and thickened at the neck of the fibula.
Only the anterior tibial group of muscles were affected, the
peroneus longus and brevis being normal. The faradic
reaction was present but reduced, in April; by July it had
completely disappeared. However, there was return of fairly
strong foot dorsiflexion by September.
Case 2.-A man, aged 42, had a sudden onset of foot-drop
while gardening in a crouching position in March, 1926. Only
the anterior tibial group of muscles were affected. There was
a history of a wound in the thigh during the war, but there
was no paralysis at that time. The duration of paralysis was
sixty days.
Case 3.-A man, aged 23, had sudden foot-drop after
kneeling for over an hour in October, 1923. It lasted for
twenty-four days.
Circumflex Nerve Paralysis
Case 4.-A woman, aged 52, discovered paralysis of the
deltoid muscle on waking one morning in February, 1936.
She was suffering from fibrositis in the scapular region following
an attack of influenza previous to the paralysis, the
duration of which was two weeks.
Case 5.-A man, aged 37, discovered paralysis of the
deltoid muscle on waking one morning in March, 1936. The
paralysis disappeared in a short time, but returned the
following day. He had been previously suffering from fibrositis
in the scapular region, and for a fortnight before the
paralysis developed cramp if sleeping on either arm. The
duration of the paralysis was only a few days.
Ulnar Nerve Paralysis
Case 6.-A lad, aged 21, experienced a sudden onset of
ulnar paralysis when riding a bicycle in April, 1932. He had
had this before, when it only lasted one day, but the duration
on the second occasion was twenty-six days. The paralysis
only affected the intrinsic hand muscles, and was apparently
due to pressure of the hand on the handle-bar.
Musculo-Spiral Nerve Paralysis
Case 7.-A man, aged 52, suddenly developed wrist-drop
after lying down for about fifteen minutes with the hand
behind the head, in February, 1926. It was a wet evening.
On examination the musculo-spinal nerve seemed to be
thickened at the middle of the posterior surface of the
humerus. The paralysis lasted for eight weeks.
AUG. 8. 1936 PARALYSIS DUE. TO POSTURE THE RITISH 2
Case 8.-A man, aged 37, had a sudden attack of wristdrop
after sleeping in a chair on a Saturday night after
indulging in alcohol, in December, 1927. The duration was
thirteen days.
Case 9.-In a man, aged 31, there was sudden onset of
wrist-drop after sleeping with one arm hanging over the side
of the bed, in January, 1928. He denied alcohol as the cause.
He returned to work after fifteen days, not completely
recovered.
Case 10.-A man, aged 37, had a sudden onset of wristdrop
after falling asleep with his head on his wrist, in
January, 1933. It lasted for seven weeks.
Case 11.-In a man, aged 68, a sudden onset of wrist-drop
occurred from the pressure of an arm over a chair in February,
1935. His teeth were very septic. It lasted for two months.
Case 12.-A man, aged 40, woke up with right wrist-drop
in June, 1930. He came to hospital five days later, when
weak active extension of the wrist had returned. No note is
available as to the duration of treatment.
In the four following cases, wrist-drop developed suddenly
without any history of pressure on the musculospiral
nerve.
Case 13.-A man, aged 59, developed wrist-drop one Sunday
morning, with pains in the scapular region, in January, 1931.
It lasted four months.
Case 14.-In a woman, aged 32, wrist-drop developed suddenly
one afternoon in June, 1q32. There was a history of
rheumatism in the shoulder one month before, but there had
been no pain immediately before the onset of the paralysis.
No note was made of the duration of treatment.
Case 15.-A lad, aged 18, had wrist-drop one evening after
work, in September, 1932. It lasted for twenty-three days.
Case 16.-In a man, aged 58, wrist-drop came on suddenly
while he was resting his elbow on the padded arm of a chair,
in November, 1929. There was thickening of the musculospiral
nerve near the external condyle of the humerus. The
duration was five weeks.
The following two cases of ulnar nerve paralysis also
occurred without any history of pressure on the nerve.
Case 17.-In a woman ulnar nerve paralysis developed suddenly
in September, 1926. The hand had been " going
numb " over the ulnar cutaneous area for some time previously.
There was thickening of the ulnar nerve above the
elbow. The duration was ten weeks.
Case 18.-In a woman, aged 58, ulnar nerve paralysis
developed suddenly after she had finished her washing, in
September, 1935. The ulnar nerve was thickened above the
elbow. The paralysis lasted for seven weeks.
These last six cases show that paralysis, presumably
from perineuritis, may develop suddenly without pressure,
and, in some cases, without any previous pain or
numbness.
Transient Paralyses
An effort was made to discover to what extent minor
cases of pressure paralysis occur which do not last a
sufficient time to require treatment. Inquiries from
hospital out-patients showed that the foot may " go
dead" on crossing the knees, and that the hand or
occasionally the whole arm may "be dead " on waking
in the morning. This " deadness clears up quickly on
movement. In many cases it only occurs occasionally,
and seems to be associated with cold and damp weather,
or fatigue. I could not obtain any history of foot-drop
or wrist-drop.
By making inquiries from people with a knowledge of
anatomy I found that:
1. Ulnar paralysis on waking in the morning, after resting
the arms on the side of a chair, or from sleeping with
the hands behind the head, is fairly common.
2. The foot may " go dead," but only rarely do the
leg muscles become definitely paralysed by sitting with the
knees crossed.
3. In a few cases the whole arm is occasionally paralysed
on waking in the morning.
These paralyses always clear up quickly on movement.
In several instances they did not occur regularly, and
were more likely to occur in damp and cold weather or
when the person was fatigued. In one case the presence
of a septic focus increased the intensity and frequency of
occurrence, but did not seem to increase the duration.
Thus the causes are similar to those of fibrositis, with
which it is often associated. I could find no case of wristdrop
or of foot-drop from kneeling.
Points of Interest
The twelve cases of pressure paralysis due to posture
represent the total number which attended the Physical
Treatment Department of King's College Hospital during
ten years. Since, presumably, all patients of this type
who attended the hospital would be ordered physical
treatment, the number seems very small, considering that
several of the postures in which paralysis occurred are
regularly assumed.
No cases of ulnar nerve paralysis from pressure on the
arm, or of foot paralysis from crossing the knees, occurred
in this series, though these two types are frequently found
in the mild form which clears up on movement. On the
other hand, I could not trace any mild cases of wrist-drop
on waking in the morning, or foot-drop after kneeling,
which recovered too quickly to require treatment, either
among hospital patients or friends.
No patient came a second time to the hospital with a
recurrence of the paralysis. This is a point against any
anatomical peculiarity being the cause.
The fact that the majority developed in the colder
months of the year suggests that cold and damp may be
a predisposing cause.
The external popliteal division of the sciatic nerve was
compressed from kneeling rather than the internal division.
Presumably the nerve must have been compressed
between the biceps tendon and the fibula.
The six wrist-drop cases were in men, and of the four
cases of wrist-drop not due to pressure only one was in a
woman. In only one case was a history of paralysis
following indulgence in alcohol obtained.
The electrical reactions varied. In one case the faradic
reaction disappeared, but in the majority it was normal
or slightly reduced, and the galvanic reaction was rather
sluggish.
Conclusion
Some of these cases can be explained as perineuritis
with added pressure. A few were probably due to pressure
only. It seems likely that in the others fatigue, cold
and damp weather, or a septic focus may have been the
predisposing cause. It would operate by affecting the
circulation of the limb and making the nerve more sensitive
to pressure without actually causing a perineuritis,
since this variation in sensitiveness to pressure is found
to be of common occurrence.
H. Vignes (Progras Med., May 30th, 1936, p. 921)
states that according to WV. H. Perkins the gravity of
spirochaetal Jaundice in pregnancy is closely xrelated to
the height and duration of the fever. Renal involvement
is a bad sign. Haemorrhages are not specially
frequent at the time of delivery. Interruption of pregnancy
is not uncommon. Experimentally Mirto observed
it in 70 per cent. of his animals. In human subjects
abortion or premature delivery may be met with, especially
in the Tropics and under unfavrourable hygienic con1-
ditions. Abortion is particularly frequent in Japan, where
severe forms of spirochaetal jaundice are common. The
foetuls as a rule is infected. No special treatment iS
indicated.

J Physiol. 2004 Oct 1;560(Pt 1):317-27. Epub 2004 Jul 29.
Human cerebral venous outflow pathway depends on posture and central venous pressure.

Gisolf J, van Lieshout JJ, van Heusden K, Pott F, Stok WJ, Karemaker JM.

Department of Physiology, Room M01-07, Academic Medical Center, University of Amsterdam, PO Box 22700, 1100 DE Amsterdam, the Netherlands. j.gisolf@amc.uva.nl.

Internal jugular veins are the major cerebral venous outflow pathway in supine humans. In upright humans the positioning of these veins above heart level causes them to collapse. An alternative cerebral outflow pathway is the vertebral venous plexus. We set out to determine the effect of posture and central venous pressure (CVP) on the distribution of cerebral outflow over the internal jugular veins and the vertebral plexus, using a mathematical model. Input to the model was a data set of beat-to-beat cerebral blood flow velocity and CVP measurements in 10 healthy subjects, during baseline rest and a Valsalva manoeuvre in the supine and standing position. The model, consisting of 2 jugular veins, each a chain of 10 units containing nonlinear resistances and capacitors, and a vertebral plexus containing a resistance, showed blood flow mainly through the internal jugular veins in the supine position, but mainly through the vertebral plexus in the upright position. A Valsalva manoeuvre while standing completely re-opened the jugular veins. Results of ultrasound imaging of the right internal jugular vein cross-sectional area at the level of the laryngeal prominence in six healthy subjects, before and during a Valsalva manoeuvre in both body positions, correlate highly with model simulation of the jugular cross-sectional area (R(2) = 0.97). The results suggest that the cerebral venous flow distribution depends on posture and CVP: in supine humans the internal jugular veins are the primary pathway. The internal jugular veins are collapsed in the standing position and blood is shunted to an alternative venous pathway, but a marked increase in CVP while standing completely re-opens the jugular veins.

A new group has been set up to discuss the merits of sleeping Inclined by raising the head of the bed six inches or 15 cm’s. Now called Inclined Therapy or I.T.

16 years of research have provided us with concrete evidence that the human body benefits from avoiding flat bed rest! Come and check us out.
Andrew K Fletcher
http://www.facebook.com/group.php?v=wall&gid=245324668896

Are we Inclined To Sit Or Not To Sit?

Sleeping with the upper half raised is pretty much useless and may cause pressure sores.

Your weight when sleeping like this is compressing your spinal column.
It is also compressing the flesh and skin on your buttocks for many hours and this may eventually lead to the development of a pressure sore.

Your circulation is only assisted partially, but compromised because the same compression of the flesh in your buttocks together with the inevitable build up of venous and arterial pressure due to the veins and arteries affected by this compression will be counter productive.

Then there is the problem with stretched tendons and ligaments, bent forward, there is inevitably some tension applied to the muscles, tendons and ligaments. Over time this can have a detrimental affect

Last but not least are the joints in the hips and in the spine, If you are sleeping on your back, this means you are bent forward for 8 hours a day. Elderly people walking bent over, unable to straighten up, was that the position they slept in?
Sleeping on your side with the upper half raised will adversely affect the hip and spine joints, sleeping on your back will also adversely affect the spine. Sleeping on your tummy will result in someone calling an ambulance..

Inclined Therapy I.T.

Sleeping on an inclined bed however, does quite the opposite.

Immediately the spine is placed under gentle traction, (the opposite to the folded posture). The weight is distributed evenly so you don't feel like a sack of potatoes when you lay down, or the bed somehow feels softer than before.

Your heart rate decreases by 10-12 beats per minute on an inclined bed while you sleep, yet manages to pump more blood around the body, which in turn causes more friction as the blood flows around faster, which in turn generates more heat and you feel warmer in bed, while the cooling system also benefits so we are able to maintain our body temperature better as more water evaporates due to the increased / maintained temperature. Sleeping flat for instance causes a 2 degree drop in body temperature around the time that most people die in bed, yet sleeping inclined does not.

Your respiration rate decreases by 4-5 breaths per minute, which is a lot, yet oxygenation improves because the lungs are inflating more and deflating more which takes a little longer so although there are less breaths per minute a greater volume of air is moved in and out, which again increases the evaporation and this in turn alters the density of the surfactant in the respiratory tract which in turn alters the density of the surfactant that is returned back into the blood and gently assists the circulation as it flows through the arteries providing we are correctly aligned / Inclined.

But we don't just have blood circulating, we have lymph and cerebrospinal fluid, we have a flow of fluids through the tissue and skin, through the bones, and even a flow through the myelin wrapped around the nervous system. The heart is not responsible for these other circulations so cannot be attributed to them. Yet we know posture and respiration plays an important roll from the literature. So could all of these independent circulations require density changes from evaporation and correct alignment with gravity to gently assist them to make the repairs required to overcome a whole range of illnesses?

I believe this to be true and indeed have already proven it many times before.

What we are already seeing unfolding here on this forum is impressive and If Foreversprings and others posts are anything to go by we are in for a very exciting 2010.

I do not believe in the majority of cases surgery is required!

Varicose veins were believed once to require surgery, yet the surgery frequently fails requiring more expensive surgery, which raises the question why?

So many people have noticed their veins no longer ache or bulge using I.T. So rather than approaching this problem by patching up the damage, why not engage the possibility that given sufficient time using I.T. we may not require surgery and those that do not respond will probably require surgery?

Interesting times are afoot.

Happy New Year

Andrew K Fletcher

Roger W. Sperry
The Nobel Prize in Physiology or Medicine 1981

In experiments with fish, frogs, and salamanders (chosen because they have great powers of regeneration), Sperry demonstrated that individual nerve fibers (which are actually different cells) behave as if each is chemically different from every other, and these chemical differences are matched in the brain. The result is that in an animal whose optic nerves are severed and then allowed to regenerate, the thousands of individual fibers that make up each optic nerve grow back into the brain and there make the same connections they had before. The animal is then able to see as if the nerves had never been severed. Proof that no adaptive reorganization of the neural circuits is involved in regeneration consisted of showing that if an eye whose optic nerve is severed is also rotated in its socket, the world seen by the eye after regeneration is still upside down and backwards. Furthermore, as in the case of the rat with the crossed nerves, no amount of retraining makes it see correctly: the animal invariably strikes to the left when it sees a worm on its right.

The conclusion that the circuitry of the brain is fixed in early development is supported by much more evidence than I can summarize here. It has given rise to a field of research focused on "axonal guidance". Sperry's result concerning the chemical individuality of each nerve fiber has been confirmed by modern molecular methods. It is a result that is loaded with meanings at many levels - from immediate consequences for neurosurgery to large and still not fully explored implications for evolution and development, and even for social-political questions. It raises other fascinating and still unsolved questions. For example, the capacity to learn obviously implies some neural plasticity. But given the basic determinism of the brain that Sperry uncovered, what does learning actually consist of at the cellular and chemical level? These and other questions posed by his findings are now being studied, and no doubt they will continue to be worked on for a long time in the future.
http://nobelprize.org/nobel_prizes/medicine/articles/sperry/index.html

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